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Case Reports in Neurological Medicine - Volume 2014 2014, Article ID 320565, 4 pages -

Case Report

Department of Neurology, Howard University Hospital, 2041 Georgia Avenue, Washington, DC 20060, USA

Department of Neurology, Howard University College of Medicine, Washington, DC 20060, USA

Received 22 July 2014; Accepted 5 October 2014; Published 28 October 2014

Academic Editor: Majaz Moonis

Copyright © 2014 Mohankumar Kurukumbi et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.


Akinetic mutism is described in various clinical presentations but typically is defined as a state wherein the patient appears awake but does not move or speak. It can be divided into two different subtypes; the most common subtypes depend on the lesion location, mesencephalic-diencephalic region, also called apathetic akinetic mutism somnolent mutism, and those involving the anterior cingulate gyrus and adjacent frontal lobes called hyperpathic akinetic mutism. The pathway of akinetic mutism is believed to originate from circuits that link the frontal and subcortical structures. This case reports a 48-year-old African American female with bilateral anterior cerebral artery stroke and akinetic mutism with coexisting thyroid storm. This patient with bilateral anterior cerebral artery infarcts presented with characteristics that are typical for akinetic mutism such as having intact eye movements but an inability to respond to auditory or visual commands. With the incidence of bilateral anterior cerebral artery ACA ischemic stroke being rare and the incidence of akinetic mutism secondary to ischemic stroke even rarer, we suspect that this patient potentially had a unilateral occlusion of anomalous anterior cerebral vasculature.

Autor: Mohankumar Kurukumbi, Thao Dang, Najeeb Crossley, Alice Esame, and Annapurni Jayam-Trouth

Fuente: https://www.hindawi.com/


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